A Homoeopathic Method for Treating Adenoid Hypertrophy - homeopathy360

A Homoeopathic Method for Treating Adenoid Hypertrophy

Abstract 

Children’s upper respiratory tract infections are a persistent global threat. The frequency of  adenoid hypertrophy and its associated problems is very high and primarily affects the  paediatric population. Whether enlarged by viral or non-infectious reasons, adenoids  undoubtedly result in feeding issues, nasal discharge, mild to severe nasal obstruction, and  associated facial traits that are easy to identify. Surgery is nearly usually required to treat  severe cases of adenoid hypertrophy, however homoeopathy can control and lessen the  problems brought on by the hypertrophy in less severe cases. This article examines  homeopathy’s function in treating adenoid hypertrophy, emphasising its holistic tenets and  customised therapeutic approaches. 

Keyword Adenoid Hypertrophy; Homoeopathy; Paediatrics; 

Introduction 

Adenoids are little tissue lumps situated above the roof of the mouth near the rear of the nose.  As a component of the immune system, they shield us from bacterial and viral illnesses. Only  children have adenoids, which begin to grow at birth and reach their maximum size between  the ages of three and five. They start to shrink around the ages of 7 and 8, and by late  adolescence, they are typically invisible, having vanished entirely by adulthood. 

Anatomy and Physiology Of Adenoids 

The adenoids, which are lymphoid tissues located beneath the mucous membrane at the  intersection of the nasopharynx’s posterior wall and roof, are essential to the immune system,  especially in the early stages of life. They have a quadrilateral form with rounded corners and  develop between weeks 12 and 14 of pregnancy. The pseudostratified ciliated columnar  epithelium lines the folds that enlarge the surface area of the adenoids. The adenoids include  lymphoid follicles made up of B cells, T cells, and dendritic cells, and this surface epithelium  is crucial for immune responses. 

These lymphoid follicles, which have crypts bordered by stratified squamous and reticulated  epithelium, are embedded in mucosal folds. Because they house lymphocytes, plasma cells, macrophages, and dendritic cells, the adenoids contribute to the first line of immunological  defence against eaten and inhaled antigens. 

Important immune cells include natural killer (NK) cells, which hunt down aberrant cells; T  cells, which can be classified as either cytotoxic or helper; and B cells, which make  antibodies. The nasal-associated lymphoid tissue (NALT) system includes the adenoids and  other lymphoid tissues in the nasopharynx. 

Etiology 

It can be generally divided into causes that are contagious and those that are not.

Infectious causes: 

  1. Viral pathogens: Adenovirus, Coronavirus, Coxsackievirus, Cytomegalovirus  (CMV), Epstein-Barr virus (EBV), Herpes simplex virus (HSV), Parainfluenza virus,  and Rhinovirus are among the viruses that can cause adenoid hypertrophy. 
  2. Bacterial pathogens: Anaerobic bacteria (such as Fusobacterium,  Peptostreptococcus, and Prevotella species) and aerobic bacteria (such as alpha-,  beta-, and gamma-hemolytic Streptococcus species, Haemophilus influenzae,  Moraxella catarrhalis, Staphylococcus aureus, Corynebacterium diphtheria, and  Mycoplasma pneumoniae) can both cause adenoid hypertrophy. 

Non-infectious causes 

  • Gastroesophageal reflux: The adenoid tissue may get irritated by acid reflux. Allergies: Hypertrophy and inflammation can result from allergic responses. Cigarette smoke exposure: Smoke exposure can lead to long-term adenoidal  irritation and hypertrophy. 
  • Adenoid hypertrophy in adults may be linked to more severe illnesses including  lymphoma, HIV infection, or sino-nasal cancer. 

Epidemiology 

Because the adenoids naturally shrink and retreat during puberty, adenoid hypertrophy is  more common in children than in adults. A recent meta-analysis found that about 34.46% of  children and adolescents have adenoid hypertrophy. The clinical and radiological results  showed a prevalence of 83.87% and 79.03%, respectively. 

Clinical Features 

The signs and symptoms of hypertrophied adenoids can be divided into two groups:  infection-related and hypertrophy-related. 

Hypertrophy-related symptoms 

In Infants: 

feeding difficulties brought on by sporadic breathing interruptions. 

Failure to thrive, increased exhaustion, and inadequate food intake 

breathing loudly and making a frothy, wet nasal sound. 

In Older Children: 

  • Nasal obstruction leading to mouth breathing.
  • Children of all ages, from neonates to teenagers, can develop obstructive sleep apnoea, which affects 1–4% of them. Its prevalence peaks between the ages of 2 and 8. 
  • Voice changes to a nasal tone.
  • subsequent chronic rhinitis and mechanical obstruction-induced nasal discharge.

Symptoms due to Infection 

  1. Nasal Discharge sinusitis and rhinitis with purulent discharge. 2. Epistaxisinfections that cause nosebleeds. 
  2. Throat Issues: Frequent upper respiratory tract infections, such as cough,  tonsillitis, pharyngitis, and post-nasal discharge.
  3. Ear Problems: Chronic otitis media, acute otitis media, and recurrent  inflammation of the Eustachian tube. The youngster is at risk for otitis media due to mechanical obstruction caused by the adenoids in the nasopharynx being close to the middle ear and the eustachian tube. Otitis media and eustachian tube dysfunction are  caused by local inflammation and mucosal oedema brought on by the regional spread  of a bacterial biofilm. 
  4. Lymphadenitis: higher deep cervical lymph node infection. 
  5. Respiratory Issues: worsening of bronchitis and bronchial asthma. 7. 

General Symptoms: Nocturnal enuresis (bedwetting) and night terrors due to  suffocation. 

Diagnosis 

Since the adenoid tissue is located in an area that is difficult to see, the diagnosis of adenoid  hypertrophy is based more on clinical characteristics than examination results. Children  typically exhibit halitosis, a persistent cough, mucopurulent posterior nasal drip, and a stuffy  nose. When hyperplastic adenoids intrude on the posterior nasal choanae, causing a buildup  of secretions in the nose, they develop a nasal intonation of speech and sometimes snort. 

  1. X-ray imaging A 25%, 50%, or 75% reduction in the amount of air column  available in front of the adenoids is categorised as mild, moderate, or severe adenoid hypertrophy, respectively. The basic plain lateral X-ray of the neck is still a great tool for screening and identifying the adenoids. 
  2. CT ScanIn addition to giving a better picture of the nasopharynx and adenoids, a  CT scan can identify the kind and nature of lesions that may be causing bone damage, which could indicate the presence of a malignant tumour. A CT scan is also helpful in the diagnosis of chronic sinusitis. 
  3. NasopharyngoscopyThe degree of adenoid hypertrophy can be more accurately  estimated by examining the adenoids with a nasopharyngoscopy. The most severe condition, where the adenoid tissue is in direct contact with the palate at rest (Grade 4), is ranked from Grade 1, where the adenoids are not in contact with other  structures.
  4. Finding the adenoid-to-nasopharyngeal (AN) ratio is the most accurate method. It is the ratio of the nasopharyngeal aperture (measured by the distance between the posterior edge of the hard palate and the spheno basiocciput) to the measurement of  the adenoid tissue (measured by the distance between the basiocciput region and the  most convex area of the adenoid pad). 

Differential Diagnosis 

  • Endonasal Foreign Bodies: Because younger children are more likely to put  objects in their noses, these can result in nasal blockage, discharge, and occasionally infection.
  • Incomplete Choanal Atresia: a congenital disorder that causes obstruction of  the choanae, the channel at the back of the nose, which can cause nasal congestion in older children and respiratory discomfort in infants.
  • Infectious or Allergic Rhinitis: Because of the inflammation and irritation of  the nasal mucosa, these disorders result in nasal congestion, discharge, and sneezing. • 

Neoplasms: Consideration should be given to both benign and malignant tumours,  especially in patients whose symptoms do not go away after therapy. 

Homoeopathic Management 

  • Baryta Carb

Chronic tonsil hypertrophy and suppuration following a mild cold or after suppressing  perspiration on the feet. Particularly appropriate for elderly individuals with dwarfism,  scrofulous children, and those with acute or chronic inflammations from colds. Increased  infiltration is more likely to result from the inflammation. 

  • Calcarea Carb

tonsil swelling and trouble swallowing. stopping of the nose when you wake up in the  morning; offensive nasal odour, similar to that of eggs; gets cold once the weather changes; 

  • Bacillinum

persistent propensity to get chilled; suggested in children having a history of consumptive  family members; The neck’s glands are sensitive and swollen; Effective as a concurrent  treatment 

  • Thuja

tonsil and throat swelling. painful swallowing, particularly when it involves empty or salivary  swallowing; mucus buildup in the back of the nares; persistent catarrh following measles;  smelt in the nose like brewing beer or fish brine. 

  • Tuberculinum

All youngsters and young students who have inherited tuberculosis may be immune from  their inheritance and their resilience will be restored if they receive two doses of  tuberculinum at long intervals. It comes in 10M, 50M, and 100M potencies. The majority of  adenoids and neck tuberculous gland cases are cured by it.

References 

1) Gupta Piyush, Menon PSN, Ramji Siddarth and Lodha Rakesh, PG Textbook of  Pediatrics, Volume 2, 1st edition, ISBN: 978-93-5152-955-2, New Delhi: Jaypee  Brothers Medical Publishers (p) Ltd. 2015Ghai O.P., Essentials Pediatrics, 6th  Edition, 1 A, Narainall,, New Delhi 

2) Phatak SR. Materia Medica of Homoeopathic Medicines: Revised and Enlarged  Edition. New Delhi, India: B Jain; 2003. 

3) Boericke, W. Pocket Manual of Homoeopathic Materia Medica & Repertory (Second  Revised and Reaugmented edition). 

4) Kent JT, Lectures on Homoeopathic Materia Medica, B Jain Publishers 5) John Henry Clarke, A Dictionary of Practical Materia Medica, 22nd edition, 

Author 

Dr. Prasoon Choudhary Professor (HOD) 

Dr. Kamal Nainawat PG Scholar, 

Department of Paediatrics, Dr. MPK Homoeopathic Medical College, Hospital  and Research Centre, Homoeopathy University, Jaipur (Rajasthan)

About the author

Dr. Kamal Nainawat

Dr. Kamal Nainawat - P.G Schooler, Dr. M.P.K Homeopathy College and Research Center, Homeopathy University, Sanganer, Jaipur